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Вступление
1. К. М. Adams, W. S. Butsch, and М. Kohlmeier, “The State of Nutrition Education at US Medical Schools,” Journal of Biomedical Education, vol. 2015 (January 2015), Article ID 357627, 7 pages. DOI: 10.1155/2015/357627.
2. “Cancer Facts & Figures 2016,” American Cancer Society, Atlanta, Georgia, 2016, http://www.cancer.Org/acs/groups/content/@research/documents/document/ acspc-047079.pdf, accessed 12/2/16.
3. “Global Cancer Facts 8c Figures, 3rd Edition,” American Cancer Society, Atlanta, Georgia, 2015, http://www.cancer.org/acs/groups/content/@research/documents/ document/acspc-044738.pdf, accessed 12/2/16.
4. N. Howlader et al. (eds.), “SEER Cancer Statistics Review, 1975–2013,” National Cancer Institute, Bethesda, MD, April 2016, http://seer.cancer.gov/csr/1975_2013/, accessed 12/2/16.
5. M. Harper, “David Graham on the Vioxx Verdict,” Forbes.com, August 19, 2005, http://www.forbes.com/2005/08/19/merck-vioxx-graham_cx_mh_0819graham. html, accessed 12/2/16.
Глава 1
1. N. Lane, Power, Sex, Suicide: Mitochondria and the Meaning of Life (New York: Oxford University Press, 2006), 3.
2. Там же.
3. Там же, location 5926.
4. “Our Best Days Are Yours,” Kelloggs, https://www.kelloggs.com/en_US/who-we-are/our-history.html, accessed 12/2/16.
5. L. B. Wrenn, Cinderella of the New South (Knoxville, TN: University of Tennessee Press, 1995), 84.
6. T. G. Graham and D. Ramsey, The Happiness Diet (New York: Rodale Books, 2012), 25.
7. R G. Mather, “Waste Products: Cotton-Seed Oil,” Popular Science Monthly, May 1894, 104.
8. Graham and Ramsey, The Happiness Diet. “Our Heritage,” Crisco, http://www.cri-sco.com/about_crisco/history.aspx, accessed 12/2/16.
9. S. Gokhale, “Marketing Crisco,” Weston A. Price Foundation, June 25, 2013, http:// www.westonaprice.org/health-topics/marketing-crisco/, accessed 12/2/16.
10. Graham and Ramsey, The Happiness Diet. T. L. Blasbalg et al., “Changes in Consumption of Omega-3 and Omega-6 Fatty Acids in the United States During the 20th Century,” American Journal of Clinical Nutrition, 93, no. 5 (May 2011): 950–962: DOI: 10.3945/ajcn. 110.006643. Epub 2011 Mar 2.
11. S. F. Halabi, Food and Drug Regulation in an Era of Globalized Markets (Cambridge, MA: Academic Press, 2015), 148.
12. T. Neltner, M. Maffini, “Generally Recognized as Secret: Chemicals Added to Food in the United States,” National Resources Defense Council, April 2014, https:// www.nrdc.org/sites/default/files/safety-loophole-for-chemicals-in-food-report.pdf, accessed 12/2/16.
13. R. J. de Souza et al., “Intake of Saturated and Trans Unsaturated Fatty Acids and Risk of All Cause Mortality, Cardiovascular Disease, and Type 2 Diabetes: Systematic Review and Meta-analysis of Observational Studies,” BMJ (2015): 351, DOI: 10.1136/bmj.h3978.
14. V. T. Samuel, K. F. Petersen, and G. I. Shulman, “Lipid-induced Insulin Resistance: Unraveling the Mechanism,” Lancet, 375 (2010): 2267–2277, DOI: 10.1016/S0140-6736(10)60408-4.
15. K. Kavanagh et al., “Trans Fat Diet Induces Abdominal Obesity and Changes in Insulin Sensitivity in Monkeys,” Obesity, 15, no. 7 (July 2007): 1675–1684, DOI: 10.1038/oby.2007.200.
16. M. C. Morris et al., “Dietary fats and the risk of incident Alzheimer’s disease,” Archives of Neurology, 60, no. 2 (2003): 194–200, DOI: 10.1001/archneur.60.2.194.
17. С. M. Benbrook, “Impacts of Genetically Engineered Crops on Pesticide Use in the U.S. – the First Sixteen Years,” Environmental Sciences Europe, 24, no. 1 (2012): 24, DOI: 10.1186/2190-4715-24-24.
18. N. Defarge et al., “Co-Formulants in Glyphosate-Based Herbicides Disrupt Aromatase Activity in Human Cells below Toxic Levels,” International Journal of Environmental Research and Public Health, 13, no. 3 (2016): 264, DOI: 10.3390/ ijerphl3030264.
19. A. Keys, “Mediterranean Diet and Public Health: Personal Reflections,” American Journal of Clinical Nutrition, 61, no. 6 supplement (1995): 1321S-1323S.
20. A. Keys, “Atherosclerosis: A Problem in Newer Public Health,” Journal ofMt. Sinai Hospital, New York, 20, no. 2 (July-August 1953): 134.
21. N. Teichholz, The Big Fat Surprise (New York: Simon & Schuster, 2014), 32–33.
22. Central Committee for Medical and Community Program of the American Heart Association, “Dietary Fat and Its Relation to Heart Attacks and Strokes,” Circulation 23 (1961): 133–136. http://circ.ahajournals.org/content/circulationa-ha/23/1/133.full.pdf, accessed 12/2/16.
23. H. M. Marvin, 1924–1964: The 40 Year War on Heart Disease (New York: American Heart Association, 1964).
24. A. Keys, “Coronary Heart Disease in Seven Countries,” Circulation, 41, no. 1 (1970): 1186–1195.
25. Dietary Guidelines Advisory Committee, “History of the Dietary Guidelines for Americans,” Nutrition and Health: Dietary Guidelines for
Americans, 2005, U.S. Department of Health and Human Services, https://health.gov/dietaryguidelines/dga2005/report/html/G5_History.htm, accessed 12/2/16.
26. Z. Harcombe et al., “Evidence from Randomised Controlled Trials Did Not Support the Introduction of Dietary Fat Guidelines in 1977 and 1983: A Systematic Review and Meta-analysis,” Open Heart, 2, no. 1 (2015): DOI: 10.1136/ openhrt-2014-000196.
27. U.S. Department of Health and Human Services and U.S. Department of Agriculture, “Key Recommendations: Components of Healthy Eating Patterns,” 2015–2020 Dietary Guidelines for Americans, 8th Edition (December 2015): 15, https:// health.gov/dietaryguidelines/2015/guidelines/chapter-1/key-recommendations/#-footnote-4, accessed 12/2/16.
28. Centers for Disease Control and Prevention, Division of Diabetes Translation, “Long-term Trends in Diabetes,” (2016), https://www.cdc.gov/diabetes/statistics/ slides/long_term_trends.pdf.
29. C. D. Fryar, M. Carroll, and C. Ogden, Division of Health and Nutrition Examination Surveys, “Prevalence of Overweight, Obesity, and Extreme Obesity Among Adults Aged 20 and Over: United States, 1960–1962 Through 2013–2014,” table 1, Centers for Disease Control and Prevention, http://www.cdc. gov/nchs/data/hestat/obesity_adult_13_14/obesity_adult_13_14.htm#Figure, accessed 12/2/16.
30. N. Howlader et al. (eds.), “SEER Cancer Statistics Review, 1975–2013.”
31. “SEER Stat Fact Sheets: Cancer of Any Site,” National Cancer Institute, http://seer. cancer.gov/statfacts/html/all.html, accessed November 28, 2016.
32. R A. Heidenreich et al., “Forecasting the Future of Cardiovascular Disease in the United States,” Circulation, 123, no. 8 (2011): 933–944, DOI: 10.1161/ CIR.0b013e31820a55f5.
33. P. Leren, “The Effect of Plasma-Cholesterol-Lowering Diet in Male Survivors of Myocardial Infarction: A Controlled Clinical Trial,” Bulletin of the New York Academy of Medicine, 44, no. 8 (1968): 1012–1020.
34. S. Dayton et al., “A Controlled Clinical Trial of a Diet High in Unsaturated Fat in Preventing Complications of Atherosclerosis,” Circulation, 40 (1969): II-1-11-63, DOI: 10.1161/01.CIR.40.1S2.II-1.
35. I. D. Frantz et al, “Test of effect of lipid lowering by diet on cardiovascular risk. The Minnesota Coronary Survey,” Arteriosclerosis, 9, no. 1 (January-February 1989): 129–135, DOI: 10.1161/01.ATV.9.1.129.
36. O. Turpeinen et al., “Dietary Prevention of Coronary Heart Disease: The Finnish Mental Hospital Study,” International Journal of Epidemiology, 9, no. 2 (1979): 99-118, DOI: 10.1093/ije/8.2.99.
37. “Controlled Trial of Soya-Bean Oil in Myocardial Infarction,” The Lancet, 292, no. 7570 (1968): 693–700, DOI: 10.1016/S0140-6736(68)90746-0.
38. “Multiple Risk Factor Intervention Trial Group: Public Annual Report, Multiple Risk Factor Intervention Trial, June 30, 1975 to July 1, 1976,” Journal of the American Medical Association, 248, no. 12 (1982): 1465–1477, https://clinicaltrials.gov/ ct2/show/NCT00000487, accessed 12/2/16.
39. P. W. Siri-Tarino et al, “Meta-analysis of Prospective Cohort Studies Evaluating the Association of Saturated Fat with Cardiovascular Disease,” American Journal of Clinical Nutrition, 91, no. 3 (2010): 535–546, DOI: 10.3945/ajcn.2009.27725.
40. R. Chowdhury et al., “Association of Dietary, Circulating, and Supplement Fatty Acids With Coronary Risk: A Systematic Review and Meta-analysis,” Annals of Internal Medicine, 160 (2014): 398–406, DOI, 10.7326/M13-1788.
41. De Souza et al., “Intake of Saturated and Trans Unsaturated Fatty Acids and Risk of 11 Cause Mortality, Cardiovascular Disease, and Type 2 Diabetes.”
42. С. E. Ramsden et al., “Use of Dietary Linoleic Acid for Secondary Prevention of Coronary Heart Disease and Death: Evaluation of Recovered Data From the Sydney Diet Heart Study and Updated Meta-analysis,” BMJ,346 (2013): DOI: 0.1136/bmj.e8707.
43. Там же.
44. M. A. Austin et al., “Low-Density Lipoprotein Subclass Patterns and Risk of Myocardial Infarction,” Journal of the American Medical Association, 260, no. 13 (1988): 1917–1921, DOI: 10.1001/jama.l988.03410130125037.
45. D. M. Dreon et al, “Change in Dietary Saturated Fat Intake Is Correlated with Change in Mass of Large Low-Density-Lipoprotein Particles in Men” American Journal of Clinical Nutrition, 67, no. 5 (1998): 828–836, accessed 12/2/16.
46. K. Gunnars, “Saturated Fat, Good or Bad?” Authority Nutrition, https://authority-nutrition.com/saturated-fat-good-or-bad/, accessed 12/2/16.
47. P. W. Siri-Tarino et al., “Saturated Fat, Carbohydrate, and Cardiovascular Disease,” American Journal of Clinical Nutrition, 91, no. 3 (2010): 502–509, DOI: 10.3945/ ajcn.2008.26285.
Глава 2
1. L. Cordain, “The Nutritional Characteristics of a Contemporary Diet Based Upon Paleolithic Food Groups,” Journal of the American Nutraceutical Association, 5, no. 5 (2002): 15–24.
2. J. J. Meidenbauer, P. Mukherjee, and T. N. Seyfried, “The Glucose Ketone Index Calculator: A Simple Tool to Monitor Therapeutic Efficacy for Metabolic Management of Brain Cancer,” Nutrition & Metabolism, vol. 12 (2015): 12. DOI: 10.1186/ sl2986-015-0009-2.
3. R. Agrawal and F. Gomez-Pinilla, ‘“Metabolic Syndrome in the Brain: Deficiency in Omega-3 Fatty Acid Exacerbates Dysfunctions in Insulin Receptor Signalling and Cognition,” The Journal of Physiology, 590, no. 10 (2012): 2485, DOI: 10.1113/ jphysiol.2012.230078.
4. J. R. Ifland et al., “Refined Food Addiction: A Classic Substance Use Disorder,” Medical Hypotheses, 72, no. 5 (May 2009): 518–526, DOI: 10.1016/j.mehy.2008.11.035.
5. T. R. Nansel et al, “Greater Food Reward Sensitivity Is Associated with More Frequent Intake of Discretionary Foods in a Nationally Representative Sample of Young Adults,” Frontiers in Nutrition, 3, no. 33, 8/18/2016, DOI: 10.3389/fnut.2016.00033.
6. S. D. Phinney and J. S. Volek, The Art and Science of Low-Carbohydrate Living (Miami, FL: Beyond Obesity LLC, 2011), 10.
7. G. D. Maurer, et al., “Differential Utilization of Ketone Bodies by Neurons and Glioma Cell Lines: a Rationale for Ketogenic Diet as Experimental Glioma Therapy,” BMC Cancer 11 (2011): 315, DOI: 10.1186/1471-2407-11-315.
8. R. Sender, S. Fuchs, and R. Milo, “Revised Estimates for the Number of Human and Bacteria Cells in the Body,” PLoS Biology, 14, no. 8 (2016): el002533, DOI: 10.1371/journal.pbio. 1002533.
9. R. Rosedale, “Life, Death, Food and the Disease of Aging,” presented at the American Academy of Anti-Aging in Orlando, Florida, 2011.
10. С. Е. Forsythe et al., “Comparison of Low Fat and Low Carbohydrate Diets on Circulation Fatty Acid Composition and Markers of Inflammation” Lipids, 43, no. 1 ("2008): 65–77, DOI: 10.1007/sll745-007-3132-7.
11. S. McKenzie, “Yoshinori Ohsumi Wins Nobel Prize for Medical Research on Cells,” CNN.com, October 3, 2016, http://www.cnn.com/2016/10/03/health/ nobel-prize-201 б-physiology-medicine-yoshinori-ohsumi/, accessed 12/2/16.
12. K. J. Bough et al., “Mitochondrial Biogenesis in the Anticonvulsant Mechanism of the Ketogenic Diet,” Annals of Neurology, 60 (2006): 223–235, DOI: 10.1002/ana.20899.
13. P. J. Cox, K. Clarke, “Acute Nutritional Ketosis: Implications for Exercise Performance and Metabolism,” Extreme Physiology & Medicine, 3 (2014): 1, DOI: 10.1186/2046-7648-3-17.
14. О. E. Owen et al., “Liver and Kidney Metabolism During Prolonged Starvation,” Journal of Clinical Investigation, 48, no. 3 (1969): 574–583.
15. M. Akram, “A Focused Review of the Role of Ketone Bodies in Health and Disease,” Journal of Medicinal Food, 16, no. 11 (November 2013): 965–967, DOI: 10.1089/jmf.2012.2592.
16. Там же.
17. Phinney and Volek, The Art and Science of Low-Carbohydrate Living, 10.
18. Interview with Jeff Volek, Ph.D., http://articles.mercola.com/sites/articles/ar-chive/2016/01/31 /high-fat-low-carb-diet-benefits.aspx, accessed 12/2/16.
19. J. C. Newman and E. Verdin, “(3-hydroxybutyrate: Much More Than a Metabolite,” Diabetes Research and Clinical Practice, 106, no. 2 (2014): 173–181, DOI: 10.1016/j. diabres.2014.08.009.
20. A. Paoli et al., “Ketogenic Diet in Neuromuscular and Neurodegenerative Diseases,” BioMed Research International, 2014 (2014), DOI: 10.1155/2014/474296.
21. M. A. McNally and A. L. Hartman, “Ketone Bodies in Epilepsy,” Journal of Neurochemistry, 121, no. 1 (2012): 28–35, DOI: 10.1111/j.l471-4159.2012.07670.x.
22. J. Moore, Keto Clarity (Victory Belt Publishing, 2014), 58.
23. A. J. Brown, “Low-Carb Diets, Fasting and Euphoria: Is There a Link between Ketosis and Gamma-hydroxybutyrate (GHB)?” Medical Hypotheses, 68, no. 2 (2007): 268–271, DOI: 10.1016/j.mehy.2006.07.043.
Глава 3
1. E. L. Knight et al., “The Impact of Protein Intake on Renal Function Decline in Women with Normal Renal Function or Mild Renal Insufficiency,” Annals of Internal Medicine, 138. no. 6 (2003): 460–467, DOI: 10.7326/0003-4819-138-6-200303180-00009.
2. M. I. Frisard et al., “Effect of 6-Month Calorie Restriction on Biomarkers of Longevity, Metabolic Adaptation, and Oxidative Stress in Overweight Individuals: A Randomized Controlled Trial,” http://jamanetwork.com/journals/jama/fullarti-cle/1108368.
3. M. E. Levine et al., “Low Protein Intake Is Associated with a Major Reduction in IGF-1, Cancer, and Overall Mortality in the 65 and Younger but Not Older Population,” Cell Metabolism, 19, no. 3 (2014): 407–417, DOI: 10.1016/j. cmet.2014.02.006.
4. J. Guevara-Aguirre et al., “Growth Hormone Receptor Deficiency Is Associated With a Major Reduction in Pro-aging Signaling, Cancer and Diabetes in Humans,” Science Translational Medicine, 3, no. 70 (2011): 70, DOI: 10.1126/ scitranslmed.3001845.
5. S. I. A. Apelo and D. W. Lamming, “Rapamycin: An InhibiTOR of Aging Emerges From the Soil of Easter Island,” Journal of Gerontology, 71, no. 7 (2016): 841–849, DOI: 10.1093/gerona/glw090.
6. S. M. Solon-Biet et al., “The Ratio of Macronutrients, Not Caloric Intake, Dictates Cardiometabolic Health, Aging, and Longevity in Ad Libi-tum-Fed Mice,” Cell Metabolism, 19, no. 3 (2014): 418–430, DOI: 10.1016/j. cmet.2014.02.009.
Глава 4
1. “Ferritin: The Test,” American Association for Clinical Chemistry, https://labtest-sonline.org/understanding/analytes/ferritin/tab/test/, accessed May 9, 2016.
2. E. D. Weinberg, “The Hazards of Iron Loading,” Metallomics, 2, no. 11 (November, 2010): 732–740, DOI: 10.1039/c0mt00023j.
3. M. D. Beaton and P. C. Adams, “Treatment of Hyperferritinemia,” Annals of Hepatology, 11, no. 3 (2012): 294–300, PMID: 22481446.
4. G. Ortiz-Estrada et al., “Iron-Saturated Lactoferrin and Pathogenic Protozoa: Could This Protein Be an Iron Source for Their Parasitic Style of Life?” Future Microbiology, 7, no. 1 (2012): 149–164, DOI: 10.2217/fmb. 11.140.
5. D. }. Fleming et al., “Dietary Factors Associated with the Risk of High Iron Stores in the Elderly Framingham Heart Study Cohort,” American Journal of Clinical Nutrition, 76, no. 6 (2002): 1375–1384, PMID: 12450906.
6. T. Iwasaki et al, “Serum Ferritin Is Associated with Visceral Fat Area and Subcutaneous Fat Area,” Diabetes Care, 28, no. 10 (2005): 2486–2491, PMID: 16186284.
7. S. К. Park et al., “Association between Serum Ferritin Levels and the Incidence of Obesity in Korean Men: A Prospective Cohort Study” Endocrine Journal, 61, no. 3 (2014): 215–224, DOI: 10.1507/endocrj.EJ13-0173.
8. Там же.
9. J. M. Fernandez-Real et al., “Serum Ferritin as a Component of the Insulin Resistance Syndrome,” Diabetes Care, 21, no. 1 (1998): 62–68, DOI: 10.2337/dia-care.21.1.62.
10. J. Montonen et al., “Body Iron Stores and Risk of Type 2 Diabetes: Results from the European Prospective Investigation into Cancer and Nutrition (EPIC)-Potsdam Study,” Diabetologia, 55, no. 10 (2012): 2613–2621, DOI: 10.1007/s00125-012-2633-y.
11. J. M. Fernandez-Real, A. Lopez-Bermejo, and W. Ricart, “Iron Stores, Blood Donation, and Insulin Sensitivity and Secretion,” Clinical Chemistry, 51, no. 7 (June 2005): 1201–1205, DOI: 10.1373/clinchem.2004.046847.
12. B. J. Van Lenten et al., “Lipid-Induced Changes in Intracellular Iron Homeostasis in Vitro and in Vivo,” Journal of Clinical Investigation, 95, no. 5 (1995): 2104–2110, DOI: 10.1172/JCI117898.
13. N. Stadler, R. A. Lindner, and M. J. Davies, “Direct Detection and Quantification of Transition Metal Ions in Human Atherosclerotic Plaques: Evidence for the Presence of Elevated Levels of Iron and Copper,” Arteriosclerosis, Thrombosis, and Vascular Biology, 24 (2004): 949–954, DOI: 10.1161/01. ATV.0000124892.90999.cb.
14. W. B. Kannel et al., “Menopause and Risk of Cardiovascular Disease: The Framingham Study,” Annals of Internal Medicine, 85 (1976): 447–452, DOI: 10.7326/0003-4819-85-4-447.
15. M. A. Lovell et al., “Copper, Iron and Zinc in Alzheimer’s Disease Senile Plaques,” Journal of the Neurological Sciences, 158, no. 1 (June 11, 1998): 47–52, DOI: 10.1016/S0022-510X(98)00092-6.
16. K. Jellinger et al., “Brain Iron and Ferritin in Parkinsons and Alzheimer’s diseases,” Journal of Neural Transmission, 2 (1990): 327, DOI: 10.1007/BF02252926.
17. G. Bartzokis et al., “Brain Ferritin Iron as a Risk Factor for Age at Onset in Neuro-degenerative Diseases,” Annals of the New York Academy of Sciences, 1012 (2004): 224–236, DOI: 10.1196/annals.l306.019.
18. S. Ayton et al., “Ferritin Levels in the Cerebrospinal Fluid Predict Alzheimer’s Disease Outcomes and Are Regulated by APOE,” Nature Communications, 6 (2015): 6760, DOI: 10.1038/ncomms7760.
19. W. Z. Zhu et al., “Quantitative MR Phase-Corrected Imaging to Investigate Increased Brain Iron Deposition of Patients with Alzheimer’s Disease,” Radiology, 253 (2009): 497–504, DOI: 10.1148/radiol.2532082324.
20. A. A. Alkhateeb and J. R. Connor, “The Significance of Ferritin in Cancer: Anti-Oxidation, Inflammation and Tumorigenesis,” Biochimica et Biophysica Acta, 1836, no. 2 (Dec 2013): 245–254, DOI: 10.1016/j.bbcan.2013.07.002.
21. J. I. Wurzelmann et al, “Iron Intake and the Risk of Colorectal Cancer,” Cancer Epidemiology, Biomarkers and Prevention, 5, no. 7 (July 1, 1996): 503–507. PMID: 8827353.
22. Y. Deugnier, “Iron and Liver Cancer,” Alcohol, 30, no. 2 (2003): 145–150.
23. L. R. Zacharski et al., “Decreased Cancer Risk after Iron Reduction in Patients with Peripheral Arterial Disease: Results from a Randomized Trial,” JNCI: Journal of National Cancer Institute, 100, no. 14 (2008): 996-1002, DOI: 10.1093/jnci/ djn209.
24. L. Valenti et al., “Association between Iron Overload and Osteoporosis in Patients with Hereditary Hemochromatosis,” Osteoporosis International, 20, no. 4 (April, 2009): 549–555, DOI: 10.1007/s00198-008-0701-4.
25. “Hemochromatosis,” National Institute of Diabetes and Digestive and Kidney Disease (2016), http://www.niddk.nih.gov/health-information/ health-topics/liver-disease/hemochromatosis/Pages/facts.aspx, accessed May 9, 2016.
26. “Welcome,” Iron Disorders Institute (2016), http://www.hemochromatosis. org/#symptoms, accessed May 9, 2016.
27. “Serum Iron Test,” MedlinePlus Medical Encyclopedia (2016), https://www.nlm. nih.gov/medlineplus/ency/article/003488.htm, accessed May 9, 2016.
28. “TIBC, UIBC, and Transferrin Test: Iron Binding Capac
ity; IBC; Serum Iron Binding Capacity; Siderophilin; Total Iron Binding Capacity; Unsaturated Iron Binding Capacity,” Lab Tests
Online (2016), https://labtestsonline.org/understanding/analytes/tibc/tab/ test/, accessed May 9, 2016.
29. L. Zacharski, “Ferrotoxic Disease: The Next Great Public Health Challenge,” Clinical Chemistry, 60, no. 11 (November 2014): 1362–1364, DOI: 10.1373/ clinchem.2014.231266.
30. P. Mangan, Dumping Iron: How to Ditch This Secret Killer and Reclaim Your Health, Phalanx Press, 2016, locations 308–312.
31. Там же, locations 1353–1356.
32. Там же, locations 1609–1612.
33. Там же, locations 416–418.
34. Там же, locations 428–431.
35. Там же, locations 582–595.
Глава 5
1. С. Manisha Chandalia et al, “Beneficial Effects of High Dietary Fiber Intake in Patients with Type 2 Diabetes Mellitus,” New England Journal of Medicine, 342 (2000): 1392–1398, DOI: 10.1056/NEJM200005113421903.
2. M. Wien et al, “A Randomized 3x3 Crossover Study to Evaluate the Effect of Hass Avocado Intake on Post-ingestive Satiety, Glucose and Insulin Levels, and Subsequent Energy Intake in Overweight Adults,” Nutrition Journal, 12 (2013): 155, DOI: 10.1186/1475-2891-12-155.
3. “Potassium,” University of Maryland Medical Center, http://umm.edu/health/med-ical/altmed/supplement/potassium, accessed November 28, 2016.
4. M. E. Cogswell et al., “Sodium and Potassium Intakes among U.S. Adults: NHANES 2003–2008,” The American Journal of Clinical Nutrition, 96, no. 3 (2012): 647–657, DOI: 10.3945/ajcn.l 12.034413.
5. M. L. Dreher and A. J. Davenport, “Hass Avocado Composition and Potential-Health Effects,” Critical Reviews in Food Science and Nutrition, 53, no. 7 (2013): 738–750, DOI: 10.1080/10408398.2011.556759.
6. R. E. Kopec et al., “Avocado Consumption Enhances Human Postprandial Provitamin A Absorption and Conversion from a Novel High-(3-Carotene Tomato Sauce and from Carrots,” Journal of Nutrition, 8 (2014), DOI: 10.3945/jn.l 13.187674.
7. N. Z. Unlu et al., “Carotenoid Absorption from Salad and Salsa by Humans Is Enhanced by the Addition of Avocado or Avocado Oil,” Journal of Nutrition, 135, no. 3 (2005): 431–436.
8. E. A. Lee et al, “Targeting Mitochondria with Avocatin В Induces Selective Leukemia Cell Death,” Cancer Research, 7Ъ, no. 12 (June 15 2015): 2478–2488, DOI: 10.1158/0008-5472.CAN-14-2676.
9. M. Notarnicola et al., “Effects of Olive Oil Polyphenols on Fatty Acid Synthase Gene Expression and Activity in Human Colorectal Cancer Cells,” Genes & Nutrition, 6, no. 1 (2011): 63–69, DOI: 10.1007/sl2263-010-0177-7.
10. A. Canuelo et al., “Tyrosol, a Main Phenol Present in Extra Virgin Olive Oil, Increases Lifespan and Stress Resistance in Caenorhabditis Elegans,” Mechanisms of Ageing and Development, 133, no. 8 (2012): 563–574, DOI: 10.1016/j. mad.2012.07.004.
11. A. H. Rahmani, A. S. Albutti, and S. M. Aly, “Therapeutics Role of Olive Fruits/Oil in the Prevention of Diseases via Modulation of Anti-Oxidant, Anti-Tumour and Genetic Activity,” International Journal of Clinical and Experimental Medicine, 7, no. 4 (2014): 799–808, PMID: 24955148.
12. J. M. Fernandez-Real et al., “A Mediterranean Diet Enriched with Olive Oil Is Associated with Higher Serum Total Osteocalcin Levels in Elderly Men at High Cardiovascular Risk,” The Journal of Clinical Endocrinology and Metabolism, 97, no. 10 (2012): 3792–3798, DOI: 10.1210/jc.2012–2221.
13. O. Garcia-Martinez et al., “Phenolic Compounds in Extra Virgin Olive Oil Stimulate Human Osteoblastic Cell Proliferation,” PLoS One, 11, no. 3 (2016): e0150045, DOI: 10.1371/journal.pone.Ol 50045.
14. “Food Fraud Database,” U.S. Pharmacopeial Convention, http://www.foodfraud. org/, accessed December 6, 2016.
15. “Sardines,” The George Mateljan Foundation, http://www.whfoods.com/genpage. php?tname=foodspice8<:dbid=147, accessed November 28, 2016.
16. K. Warner, W. Timme, B. Lowell, and M. Hirshfield, “Oceana Study Reveals Seafood Fraud Nationwide,” February 2013, http://usa.oceana.org/sites/default/files/ National_Seafood_Fraud_Testing_Results_Highlights_FINAL.pdf, accessed December 8, 2016.
17. http://articles.mercola.com/sites/articles/archive/2015/05/13/seafood-shrimp-in – dustry- fraud. aspx#_edn 1.
18. http://articles.mercola.com/sites/articles/archive/2015/05/13/seafood-shrimp-industry- fraud. aspx#_edn2.
19. http://articles.mercola.com/sites/articles/archive/2015/05/13/seafood-shrimp-in – dustry-fraud.aspx#_edn3.
20. http://articles.mercola.com/sites/articles/archive/2015/05/13/seafood-shrimp-industry- fraud. aspx#_edn 15.
21. http://articles.mercola.com/sites/articles/archive/2015/05/13/seafood-shrimp-industry-fraud. aspx#_edn 16.
22. N. Greenfield, “The Smart Seafood Buying Guide,” https://www.nrdc.org/stories/ smart-seafood-buying-guide, accessed November 28, 2016.
23. M. Neuhouser et al., “Food and Nutrient Intakes, and Health: Current Status and Trends,” Dietary Guidelines Advisory Committee, https://health.gov/dietar-yguidelines/2015-BINDER/meeting7/docs/DGAC-Meeting-7-SC-l.pdf, accessed December 8, 2016.
24. B. S. Luh, W. S. Wong, and N. E. El-Shimi, “Effect of Processing on Some Chemical Constituents of Pistachio Nuts,” Journal of Food Quality, 5 (1982): 33–41, DOI: 10.111 l/j.l745-4557.1982.tb00954.x.
25. S. M. Solon-Biet et al., “The Ratio of Macronutrients, Not Caloric Intake, Dictates Cardiometabolic Health, Aging, and Longevity in Ad Libitum-Fed Mice,” Cell Metabolism, 19, no. 3 (418–430), DOI: 10.1016/j.cmet.2014.02.009.
26. A. Villalvilla et al, “Lipid Transport and Metabolism in Healthy and Оsteoarthritic Cartilage,” International Journal of Molecular Sciences, 14, no. 10 (2013): 20793-20808, DOI: 10.3390/ijmsl41020793.
Глава 6
1. J. A. Vasquez and J. E. Janosky, “Validity of Bioelectrical-Impedance Analysis in Measuring Changes in Body Mass During Weight Reduction,” American Journal of Clinical Nutrition, 54, no. 6 (1991): 970–975, PMID 1957829.
Глава 7
1. A. G. Bergqvist et al., “Fasting Versus Gradual Initiation of the Ketogenic Diet: A Prospective, Randomized Clinical Trial of Efficacy,” Epilepsia, 46, no. 11 (November 2005): 1810–1819, DOI: 10.1111/j.l528-1167.2005.00282.x.
Глава 8
1. “A Daily Walk Can Add Seven Years to Your Life,” The Independent, http://www.independent.co.uk/life-style/health-and-families/health-news/a-daily-walk-can-add-seven-year-to-your-life-10478821.html, accessed November 28, 2016.
Глава 9
1. C. Newell et al., “Ketogenic Diet Modifies the Gut Microbiota in a Murine Model of Autism Spectrum Disorder,” Molecular Autism, 7, no. 1 (2016): 37, DOI: 10.1186/sl3229-016-0099-3.
2. S. B. Eaton and M. Konner, “Paleolithic Nutrition – A Consideration of Its Nature and Current Implications,” New England Journal of Medicine, 312 (1985): 283–289, DOI: 10.1056/NEJM198501313120505.
3. D. Piovesan et al., “The Human ‘Magnesome: Detecting Magnesium Binding Sites on Human Proteins” BMC Bioinformatics, 13, no. 14 supplement (2012): S10, DOI: 10.1186/1471-2105-13-S14-S10.
4. “Magnesium: Fact Sheet for Health Professionals,” U.S. Department of Health and Human Services, https://ods.od.nih.gov/factsheets/Magnesium-HealthProfes-sional/, accessed November 28, 2016.
Глава 10
1. “Overweight and Obesity Statistics,” U.S. Department of Health and Human Services, https://www.niddk.nih.gov/health-information/health-statistics/Pages/over-weight-obesity-statistics.aspx, accessed November 28, 2016.
2. S. Gill and S. Panda, “A Smartphone App Reveals Erratic Diurnal EatingPatterns in Humans that Can Be Modulated for Health Benefits,” Cell Metabolism, 22, no. 5 (November 3, 2015): 789–798, DOI: 10.1016/j.cmet.2015.09.005.
3. “Autophagy Key to Restoring Function in Old Muscle Stem Cells,” Sens Research Foundation, https://www.fightaging.org/archives/2016/01/autophagy-key-to-restor-ing-function-in-old-muscle-stem-cells/, accessed November 28, 2016.
4. A. M. Johnstone et al., “Effect of an Acute Fast on Energy Compensation and Feeding Behaviour in Lean Men and Women,” International Journal of Obesity, 26, no 12 (2002): 1623–1628, DOI: 10.1038/sj.ijo.0802151.
5. Gill and Panda, “A Smartphone App Reveals Erratic Diurnal Eating Patterns in Humans.”
6. V. К. M. Halagappa et al, “Intermittent Fasting and Caloric Restriction Ameliorate Age-Related Behavioral Deficits in the Triple-Transgenic Mouse Model of Alzheimer’s Disease,” Neurobiology of Disease, 26, no. 1 (2007): 212–220, DOI: 10.1016/j.nbd.2006.12.019.
7. A. M. Stranahan and M. P. Mattson, “Recruiting Adaptive Cellular Stress Responses for Successful Brain Ageing,” Nature Reviews Neuroscience, 13, no. 3 (March 2012): 209–216, DOI: 10.1038/nrn3151.
8. S. Brandhorst et al., “A Periodic Diet That Mimics Fasting Promotes Multi-System Regeneration, Enhanced Cognitive Performance, and Healthspan,” Cell Metabolism, 22, no. 1 (July 7, 2015): 86–99, DOI: 10.1016/j. cmet.2015.05.012.
9. K. Varady et al, “Alternate Day Fasting for Weight Loss in Normal Weight and Overweight Subjects: A Randomized Controlled Trial,” Nutrition Journal, 12 (2013): 146, DOI: 10.1186/1475-2891-12-146.
10. I. Ahmet et al., “Chronic Alternate Day Fasting Results in Reduced Diastolic Compliance and Diminished Systolic Reserve in Rats,” Journal of Cardiac Failure, 16, no. 10 (2010): 843–853, DOI: 10.1016/j.cardfail.2010.05.007.
11. C. R. Marinac et al., “Prolonged Nightly Fasting and Breast Cancer Prognosis,” Journal of the American Medical Association Oncology, 2, no. 8 (2016): 1049–1055, DOI: 10.1001/jamaoncol.2016.0164.
12. R. Pamplona, “Mitochondrial DNA Damage and Animal Longevity: Insights from Comparative Studies,” Journal of Aging Research, 2011 (2011): DOI: 10.4061/2011/807108.
13. P. Sonksen and J. Sonksen, “Insulin: Understanding Its Action in Health and Disease,” British Journal of Anaesthesia, 85, no. 1 (2000): 69–79, DOI: 10.1093/ bja/85.1.69.
14. M. J. Wargovich and J. E. Cunningham, “Diet, Individual Responsiveness and Cancer Prevention,” The Journal of Nutrition, 133 (July 2003): 2400S-2403S, PMID 12840215.
15. M. V. Chakravarthy and F. W. Booth, “Eating, Exercise, and ‘Thrifty’ Genotypes: Connecting the Dots toward an Evolutionary Understanding of Modern Chronic Diseases,” Journal of Applied Physiology, 96, no. 1 (2004): 3-10, DOI: 10.1152/jap-plphysiol.00757.2003.
16. V. D. Longo and M. P. Mattson, “Fasting: Molecular Mechanisms and Clinical Applications,” Cell Metabolism, 19, no. 2 (2014): 181–192, DOI:10.1016/jc-met.2013.12.008.
Глава 11
1. “Body to the Earths Surface Electrons,” Journal of Environmental and Public Health, 2012 (2012), DOI: 10.1155/2012/291541.
2. J. L. Oschman, G. Chevalier, and R. Brown, “The Effects of Grounding (Earthing) on Inflammation, the Immune Response, Wound Healing, and Prevention and Treatment of Chronic Inflammatory and Autoimmune Diseases,” Journal of Inflammation Research, 8 (2015): 83–96, DOI: 10.2147/JIR.S69656.
3. D. Z. Kochan et al., “Circadian Disruption and Breast Cancer: An Epigenetic Link?” Oncotarget, 6, no. 19 (2015): 16866-16682. DOI:10.18632/oncotarget.4343.
4. M. Dunbar and R. Melton, “The Lowdown on Light: Good vs. Bad, and Its Connection to AMD,” Review of Optometry, https://www.reviewofoptometry.com/ce/ the-lowdown-on-blue-light-good-vs-bad-and-its-connection-toamd-109744, accessed November 28, 2016.
5. D. Peretti et al., “RBM3 Mediates Structural Plasticity and Protective Effects of Cooling in Neurodegeneration” Nature, 518, no. 7538 (2015): 236–239, DOI: 10.1038/nature 14142.
Приложение А
1. Load Diet in Treatment of Acne Vulgaris in Korean Patients: A Randomized, Controlled Trial.” Acta Dermato Venereologica, 92, no. 3 (May 2012): 241–246, DOI: 10.2340/00015555-1346.
2. L. Knott et al., “Regulation of Osteoarthritis by Omega-3 (n-3) Polyunsaturated Fatty Acids in a Naturally Occurring Model of Disease,” Osteoarthritis Cartilage, 19, no. 9 (September 2011): 1150–1157, DOI: 10.1016/j.joca.2011.06.005.
3. L. Cordain et al., “Acne Vulgaris: A Disease of Western Civilization,” Archives of Dermatology, 138, no. 12 (December 2002): 1584–1590, DOI: 10.1001/arch-derm.138.12.1584.
4. R. N. Smith et al., “A Low-Glycemic-Load Diet Improves Symptoms in Acne Vulgaris Patients: A Randomized Controlled Trial,” American Journal of Clinical Nutrition, 86, no. 1 (July 2007): 107–115.
5. Kwon et al., “Clinical and Histological Effect of a Low Glycaemic Load Diet in Treatment of Acne Vulgaris in Korean Patients.”
6. S. N. Mahmood and W.P. Bowe, “Diet and Acne Update: Carbohydrates Emerge as the Main Culprit,” Journal of Drugs in Dermatology, 13, no. 4 (April 2014): 428–435.
7. “2015 Alzheimer’s Disease Facts and Figures,” Alzheimer’s Association, https://www. alz.org/facts/downloads/facts_figures_2015.pdf, accessed November 28, 2016.
8. World Health Organization. “Dementia: a Public Health Priority” (Geneva, SUI: World Health Organization, 2012), PMID: 19712582.
9. B. D. James et al., “Contribution of Alzheimer Disease to Mortality in the United States,” Neurology,published online before print March 5, 2014, DOI: 10.1212/ WNL.0000000000000240.
10. V. R. Bitra, D. Rapaka, and A. Akula, “Prediabetes and Alzheimer’s Disease,” Indian Journal of Pharmaceutical Sciences, 77, no. 5 (2015): 511–514.
11. S. M. de la Monte, “Insulin Resistance and Alzheimer’s Disease,” BMB Reports, 42, no. 8 (2009): 475–481.
12. R. O. Roberts et al., “Relative Intake of Macronutrients Impacts Risk of Mild Cognitive Impairment or Dementia,” Journal of Alzheimer s Disease, 32, no. 2 (2012), 329–339. DOI: 10.3233/JAD-2012-120862.
13. S. T. Henderson et al., “Study of the Ketogenic Agent AC-1202 in Mild to Moderate Alzheimer’s Disease: A Randomized, Double-Blind, Placebo-Controlled, Multicenter Trial,” Nutrition & Metabolism, 6 (2009): 31. DOI: 10.1186/1743-7075-6-31, PMID: 19664276.
14. }. Yao and R. D. Brinton, “Targeting Mitochondrial Bioenergetics for Alzheimer’s Prevention and Treatment,” Current Pharmaceutical Design, 17, no. 31 (2011): 3474–3479, PMID: 21902662.
15. J. M. Hootman et al., “Updated Projected Prevalence of Self-Reported Doctor-Diagnosed Arthritis and Arthritis-Attributable Activity Limitation Among US Adults, 2015–2040.” Arthritis & Rheumatololgy, 68, no. 7 (July 2016): 1582–1587, DOI: 10.1002/art.39692.
16. Knott et al., “Regulation of Osteoarthritis by Omega-3 (n-3) Polyunsaturated Fatty Acids in a Naturally Occurring Model of Disease.”
17. Y. M. Bastiaansen-Jenniskens et al., “Monounsaturated and Saturated, but Not n-6 Polyunsaturated Fatty Acids Decrease Cartilage Destruction under Inflammatory Conditions: A Preliminary Study.” Cartilage, 4 no. 4 (2013), 321–328. DOI: 10.1177/1947603513494401.
18. D. N. Ruskin, M. Kawamura, and S. A. Masino, “Reduced Pain and Inflammation in Juvenile and Adult Rats Fed a Ketogenic Diet,” PLoS One, 4, no. 12 (2009): e8349, D01:10.1371/journal.pone.0008349.
19. S. A. Masino and D. N. Ruskin, “Ketogenic Diets and Pain,” Journal of Child Neurology, 28, no. 8 (2013): 993-1001. DOI: 10.1177/0883073813487595.
20. “Vital Signs: Preventable Deaths from Heart Disease & Stroke,” Centers for Disease Control and Prevention, http://www.cdc.gov/dhdsp/vital_signs.htm, accessed November 28, 2016.
21. B. Hoogwerf et al., “Blood Glucose Concentrations <125 mg/dl and Coronary Heart Disease Risk J American Journal of Cardiology, 89, no. 5, (2002): 596–599, DOI: 10.1016/S0002-9149(01)02302-5.
22. N. V Dhurandhar and D. Thomas, “The Link between Dietary Sugar Intake and Cardiovascular Disease Mortality: An Unresolved Question,” Journal of the American Medical Association, 313, no. 9 (2015): 959–960. DOI: 10.1001/jama.2014.18267, accessed 12/2/16.
23. Q. Yang et al., “Added Sugar Intake and Cardiovascular Diseases Mortality Among US Adults,” JAMA Internal Medicine, 174, no. 4 (2014), 516–524, DOI: 10.1001/ jamainternmed.2013.13563.
24. L. Schwingshackl et al., “Comparison of Effects of Long-Term Low-Fat vs High-Fat Diets on Blood Lipid Levels in Overweight or Obese Patients: A Systematic Review and Meta-Analysis.” Journal of the Academy of Nutrition and Dietetics, 113, no. 12 (2013), 1640–1661, DOI: 10.1016/j.jand.2013.07.010.
25. C. L. Gibson, A. N. Murphy, and S. P. Murphy, “Stroke Outcome in the Ketogenic State: A Systematic Review of the Animal Data,” Journal of Neurochemistry, 123, no. 2 (2012), 52–57, DOI: 10.1111/j.l471-4159.2012.07943.x.
26. “Epilepsy Fast Facts,” Centers for Disease Control and Prevention, http://www.cdc. gov/epilepsy/basics/fast-facts.htm, accessed November 28, 2016.
27. J. W. Wheless, “History of the Ketogenic Diet,” Epilepsia, 49, Suppl. 8 (November 2008): 3–5, DOI: 10.111 l/j.1528–1167.2008.01821.x.
28. K. Martin et al, “Ketogenic Diet and Other Dietary Treatments for Epilepsy,” Cochrane Database of Systematic Reviews, 2 (2016), DOI: 10.1002/14651858. CD001903.pub3.
29. “What Is Fibromyalgia?” (November 2014), http://www.niams.nih.gov/.
30. Mayo Clinic, “Diseases and Conditions: Fibromyalgia,” http://www.mayoclinic. org/diseases-conditions/fibromyalgia/basics/causes/con-20019243, accessed November 28, 2016.
31. Paper presented at the Annual Meeting of the American College of Nutrition in Orlando, Florida, October 2001.
32. M. Meeus et al., “The Role of Mitochondrial Dysfunctions Due to Oxidative and Nitrosative Stress in the Chronic Pain or Chronic Fatigue Syndromes and Fibromyalgia Patients: Peripheral and Central Mechanisms as Therapeutic Targets?” Expert Opinion on Therapeutic Target, 17, no. 9 (2013): 1081–1089, DOI: 10.1517/14728222.2013.818657.
33. A. Ernst and J. Shelley-Tremblay, “Non-Ketogenic, Low Carbohydrate Diet Predicts Lower Affective Distress, Higher Energy Levels and Decreased Fibromyalgia Symptoms in Middle-Aged Females with Fibromyalgia Syndrome as Compared to the Western Pattern Diet,” Journal of Musculoskeletal Pain, 21, no. 4 (2013): 365–370, DOI: 10.3109/10582452.2013.852649.
34. “GERD,” American Gastroenterological Association, http://www.gastro.org/pa-tient-care/conditions-diseases/gerd, accessed November 28, 2016.
35. “A Sunny Day in Pharmaland: The 2015 Pharma Report,” Medical Marketing & Media, http://media.mmm-online.com/documents/119/pharma_report_2015_29732. pdf, accessed November 28, 2016.
36. Singh et al., “Weight Loss Can Lead to Resolution of Gastroesophageal Reflux Disease Symptoms: A Prospective Intervention Trial,” Obesity, 21, no. 2 (2013), DOI: 10.1002/oby.20279.
37. G. L. Austin et al., “A Very Low-Carbohydrate Diet Improves Gastroesophageal Reflux and Its Symptoms,” Digestive Diseases and Sciences, 51, no. 8 (August 2006): 1307–1312, DOI: 10.1007/sl0620-005-9027-7.
38. Singh et al., “Weight Loss Can Lead to Resolution of Gastroesophageal Reflux Disease Symptoms.”
39. G. L. Austin et al., “A Very Low-Carbohydrate Diet Improves Symptoms and Quality of Life in Diarrhea-Predominant Irritable Bowel Syndrome,” Clinical Gastroenterology and Hepatology: The Official Clinical Practice Journal of the American Gastroenterological Association, 7, no. 6 (2009): 706–708. el. DOI: 10.1016/j. cgh.2009.02.023.
40. Z. Zheng et al., “Staple Foods Consumption and Irritable Bowel Syndrome in Japanese Adults: A Cross-Sectional Study,” PLoS One, 10, no. 3 (2015): eOl 19097, DOI: 10.1371/journal.pone.Ol 19097.
41. “Migraine Statistics,” Migraine.com, https://migraine.com/migraine-statistics/, accessed November 28, 2016.
42. PubMed.gov, https://www.ncbi.nlm.nih.gov/pubmed/?term=migraine+food-i-allergy, accessed November 28, 2016.
43. K. Alpay et al., “Diet Restriction in Migraine, Based on IgG Against Foods: A Clinical Double-blind, Randomised, Cross-over Trial,” Cephalalgia, 30, no. 7 (2010): 829–837, DOI: 10.1177/0333102410361404.
44. C. Di Lorenzo et al., “Migraine Improvement During Short Lasting Ketogene-sis: A Proof-of-Concept Study,” European Journal of Neurology, 22, no. 1 (2015): 170–177, DOI: 10.1111/ene.l2550.
45. C. Di Lorenzo et al., “Diet Transiently Improves Migraine in Two Twin Sisters: Possible Role of Ketogenesis?” Functional Neurology, 28, no. 4 (2013): 305–308.
46. K. L. Munger et al., “Vitamin D Intake and Incidence of Multiple Sclerosis,” Neurology, 62, no. 1, (2004): 60–65, PMID: 14718698.
47. D. Y. Kim et al., “Inflammation-Mediated Memory Dysfunction and Effects of a Ketogenic Diet in a Murine Model of Multiple Sclerosis,” PLoS One, 7, no. 5 (2012): e35476, DOI: 10.1371/journal.pone.0035476.
48. M. Storoni and G. T. Plant, “The Therapeutic Potential of the Ketogenic Diet in Treating Progressive Multiple Sclerosis,” Multiple Sclerosis International, 2015 (2015): 681289, DOI: 10.1155/2015/681289.
49. Там же.
50. “Non-Alcoholic Fatty Liver Disease,” American Liver Foundation, http://www.liv-erfoundation.org/abouttheliver/info/nafld/, accessed November 28, 2016.
51. S. S. Sundaram, “Pediatric Non-Alcoholic Fatty Liver Disease,” American Liver Foundation, http://www.liverfoundation.org/chapters/rockymountain/doctorsnotes/ pediatricnafld/, accessed November 28, 2016.
52. J. Ma et al., “Sugar-sweetened Beverage, Diet Soda, and Fatty Liver Disease in the Framingham Heart Study Cohorts,” Journal of Hepatology, 63, no. 2 (2015): 462–469, DOI: 10.1016/j.jhep.2015.03.032.
53. J. D. Browning et al., “Short-term Weight Loss and Hepatic Triglyceride Reduction: Evidence of a Metabolic Advantage with Dietary Carbohydrate Restriction,” The American Journal of Clinical Nutrition, 93, no. 5 (2011): 1048–1052. DOI: 10.3945/ajcn.l 10.007674.
54. J. Perez-Guisado and A. Munoz-Serrano, “The Effect of the Spanish Ketogenic Mediterranean Diet on Nonalcoholic Fatty Liver Disease: A Pilot Study,” Journal of Medicinal Food, 14, no. 7–8 (July-August 2011): 677–680, DOI: 10.1089/ jmf.2011.0075.
55. D. Tendler et al., “The Effect of a Low-Carbohydrate, Ketogenic Diet on Nonalcoholic Fatty Liver Disease: A Pilot Study,” Digestive Diseases and Sciences, 52, no. 2 (February, 2007): 589–593, DOI: 10.1007/sl0620-006-9433-5.
56. P. Kennedy, “The Fat Drug.” The New York Times, March 8, 2014, http://www.ny-times.com/2014/03/09/opinion/sunday/the-fat-drug.html?_r=0, accessed 12/2/16.
57. H.-Y. Kim et al., “Phosphatidylserine-dependent Neuroprotective Signaling Promoted by Docosahexaenoic Acid,” Prostaglandins, Leukotrienes, and Essential Fatty Acids, 82, no. 4–6 (2010): 165–172, DOI: 10.1016/j.plefa.2010.02.025.
58. H.-Y. Kim et al., “N-Docosahexaenoylethanolamide Promotes Development of Hippocampal Neurons,” The Biochemical Journal, 435, no. 2 (2011): 327–336, DOI: 10.1042/BJ20102118.
59. R. Palacios-Pelaez, W. J. Lukiw, and N. G. Bazan, “Omega-3 Essential Fatty Acids Modulate Initiation and Progression of Neurodegenerative Disease,” Molecular Neurobiology, 41, no. 2–3 (June 2010): 367–374, DOI: 10.1007/sl2035-010-8139-z.
60. Interview with J. J. Virgin, http://articles.mercola.com/sites/articles/ar-chive/2014/02/09/fish-oil-brain-health.aspx, accessed 12/2/16.
61. S. Smith, “Fish Oil Helped Save Our Son,” CNN, http://www.cnn.com/2012/10/19/ health/fish-oil-brain-injuries/index.html, accessed 12/2/16.
62. M. L. Prins and J. H. Matsumoto, “The Collective Therapeutic Potentialof Cerebral Ketone Metabolism in Traumatic Brain Injury,” Journal of Lipid Research, 55, no. 12 (2014): 2450–2457, DOI: 10.1194/jlr.R046706.
63. H. Algattas and J. H. Huang, “Traumatic Brain Injury Pathophysiology and Treatments: Early, Intermediate, and Late Phases Post-Injury,” International Journal of Molecular Sciences, 15, no. 1 (2014): 309–341, DOI: 10.3390/ijmsl5010309.
64. Там же.
65. M. L. Prins, L. S. Fujima, and D. A. Hovda, “Age-dependent Reduction of Cortical Contusion Volume by Ketones After Traumatic Brain Injury,” Journal of Neuroscience Research, 82, no. 3 (November 1, 2005): 413–420, DOI: 10.1002/jnr.20633.
66. Z. G. Hu et al., “The Protective Effect of the Ketogenic Diet on Traumatic Brain Injury-Induced Cell Death in Juvenile Rats,” Brain Injury, 23, no. 5 (2009): 459–465, DOI: 10.1080/02699050902788469.
67. “National Diabetes Statistics Report, 2014,” National Center for Chronic Disease Prevention and Health Promotion, http://www.cdc.gov/diabetes/pubs/statsre-portl4/national-diabetes-report-web.pdf, accessed 12/2/16.
68. “Diabetes Facts and Figures,” International Diabetes Foundation, http://www.idf. org/about-diabetes/facts-figures, accessed November 28, 2016.
69. D. Dabelea et al., “Prevalence of Type 1 and Type 2 Diabetes Among Children and Adolescents From 2001 to 2009,” Journal of the American Medical Association, 311, no. 17 (2014): 1778–1786, DOI: 10.1001/jama.2014.3201.
70. S. Vijan et al., “Effect of Patients’ Risks and Preferences on Health Gains with Glucose Lowering in Type 2 Diabetes,” JAMA Internal Medicine, 174, no. 8 (2014): 1227–1234, DOI: 10.1001/jamainternmed.2014.2894.
71. M. M. Poplawski et al., “Reversal of Diabetic Nephropathy by a Ketogenic Diet,” PLoS One, 6, no. 4 (2011): el8604, DOI: 10.1371/journal.pone.0018604.
72. R. D. Feinman et al., “Dietary Carbohydrate Restriction as the First Approach in Diabetes Management: Critical Review and Evidence Base,” Nutrition, 31, no. 1 (2015): 1-13, DOI: 10.1016/j.nut.2014.06.01.1.
73. “Making Healthy Food Choices: Grains and Starchy Vegetables,” American Diabetes Association, http://www.diabetes.org/food-and-fitness/food/what-can-i-eat/ making-healthy-food-choices/grains-and-starchy-vegetables.html, accessed November 28, 2016.
Приложение Б
1. M. S. Touillaud et al., “Dietary Lignan Intake and Postmenopausal Breast Cancer Risk by Estrogen and Progesterone Receptor Status,” Journal of the National Cancer Institute, 2007, 99(6): 475–486, DOI: 10.1093/jnci/djk096.
2. A. Ahmad et al., “A Review on Therapeutic Potential of Nigella Sativa: A Miracle Herb,” Asian Pacific Journal of Tropical Biomedicine, 2013, 3(5): 337–352, DOI: 1016/S2221-1691(13)60075-1.
3. S. Hasani-Ranjbar, Z. Jouyandeh, and M. A. Abdollahi, “A Systematic Review of Anti-Obesity Medicinal Plants – An Update,” Journal of Diabetes and Metabolic Disorders, 2013, 12:28, DOI: 10.1186/2251-6581-12-28.
4. M. Yadav et al., ’’Medicinal and biological Potential of Pumpkin: An Updated Review,” Nutrition Research Reviews, 2010, 23(2), 184–190, DOI: 10.1017/ S0954422410000107.
5. W. A. Morgan and B. J. Clayshulte, “Pecans Lower Low Density Lipoprotein Cholesterol in People with Normal Lipid Levels,” Journal of the American Dietetic Association, March 2000, 100(3): 312–318, DOI: 10.1016/S0002-8223(00)00097-3.
6 Oakridge Associated Universities, “Brazil Nuts,” http://www.orau.org/PTP/collec-tion/consumer%20products/brazilnuts.htm.
